recombinant mouse fibroblast growth factors fgf basic protein (R&D Systems)
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Recombinant Mouse Fibroblast Growth Factors Fgf Basic Protein, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/recombinant+mouse+basic+fgf/Recombinant+Mouse+FGF-10+Protein/pmc12978157-73-27-35
Average 94 stars, based on 26 article reviews
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Suspension:Article Title: Prominin‐1 controls stem cell activation by orchestrating ciliary dynamics Article Snippet: Explants were incubated in 1% type I collagenase (Sigma‐Aldrich) in HBSS with 1% penicillin–streptomycin and 1% Fungizone ® Antimycotic (all from Thermo Fisher Scientific) for 60 min and dissociated by pipetting 20 times every 15 min. Collagenase was deactivated with equal amount of DMEM containing 10% fetal bovine serum (Sigma‐Aldrich) and 1% penicillin–streptomycin. .. The cell suspension was then centrifuged at 1,000 × g for 5 min, and the resulting pellet was re‐suspended in DMEM/F12 (Thermo Fisher Scientific) containing B27 supplements (Minus vitamin A version, Thermo Fisher Scientific), recombinant mouse EGF (20 ng/ml final concentration; carrier‐free form, R&D Systems), Article Title: Posttreatment with dexmedetomidine aggravates LPS-induced myocardial dysfunction partly via activating cardiac endothelial α 2A -AR in mice. Article Snippet: Background: Dexmedetomidine (DEX) administered before or at 30 min after sepsis induction was reported to alleviate septic cardiomyopathy in experimental models.. However, sepsis is a life-threatening organ dysfunction due to infection-induced dysregulated host response, whether DEX treatment in the presence of organ dysfunction affects septic cardiomyopathy is unknown.. This study investigated the effect of DEX posttreatment on septic cardiomyopathy. Recombinant:Article Title: Prominin‐1 controls stem cell activation by orchestrating ciliary dynamics Article Snippet: Explants were incubated in 1% type I collagenase (Sigma‐Aldrich) in HBSS with 1% penicillin–streptomycin and 1% Fungizone ® Antimycotic (all from Thermo Fisher Scientific) for 60 min and dissociated by pipetting 20 times every 15 min. Collagenase was deactivated with equal amount of DMEM containing 10% fetal bovine serum (Sigma‐Aldrich) and 1% penicillin–streptomycin. .. The cell suspension was then centrifuged at 1,000 × g for 5 min, and the resulting pellet was re‐suspended in DMEM/F12 (Thermo Fisher Scientific) containing B27 supplements (Minus vitamin A version, Thermo Fisher Scientific), recombinant mouse EGF (20 ng/ml final concentration; carrier‐free form, R&D Systems), Article Title: Isolation, Characterization, and Transplantation of Cardiac Endothelial Cells Article Snippet: Mononuclear cells both acquired directly from the heart (unsorted cells, US) and sorted by flow cytometer (sorted cells, S) were resuspended in 5 mL growth media and seeded on uncoated 100 × 20 mm of tissue culture dishes (BD bioscience) at 37°C, 5% CO 2 , 5% O 2 in a humidified incubator. .. Growth media contained high glucose Dulbecco's Modified Eagle's Medium (DMEM) supplemented with 10% USDA Tested fetal bovine serum, 100 units/mL Penicillin, 100 μ g/mL Streptomycin (all from HyClone), 10 ng/mL recombinant mouse VEGF (R&D, Minneapolis, MN) and 20 ng/mL Article Title: Posttreatment with dexmedetomidine aggravates LPS-induced myocardial dysfunction partly via activating cardiac endothelial α 2A -AR in mice. Article Snippet: Background: Dexmedetomidine (DEX) administered before or at 30 min after sepsis induction was reported to alleviate septic cardiomyopathy in experimental models.. However, sepsis is a life-threatening organ dysfunction due to infection-induced dysregulated host response, whether DEX treatment in the presence of organ dysfunction affects septic cardiomyopathy is unknown.. This study investigated the effect of DEX posttreatment on septic cardiomyopathy. Concentration Assay:Article Title: Prominin‐1 controls stem cell activation by orchestrating ciliary dynamics Article Snippet: Explants were incubated in 1% type I collagenase (Sigma‐Aldrich) in HBSS with 1% penicillin–streptomycin and 1% Fungizone ® Antimycotic (all from Thermo Fisher Scientific) for 60 min and dissociated by pipetting 20 times every 15 min. Collagenase was deactivated with equal amount of DMEM containing 10% fetal bovine serum (Sigma‐Aldrich) and 1% penicillin–streptomycin. .. The cell suspension was then centrifuged at 1,000 × g for 5 min, and the resulting pellet was re‐suspended in DMEM/F12 (Thermo Fisher Scientific) containing B27 supplements (Minus vitamin A version, Thermo Fisher Scientific), recombinant mouse EGF (20 ng/ml final concentration; carrier‐free form, R&D Systems), Modification:Article Title: Isolation, Characterization, and Transplantation of Cardiac Endothelial Cells Article Snippet: Mononuclear cells both acquired directly from the heart (unsorted cells, US) and sorted by flow cytometer (sorted cells, S) were resuspended in 5 mL growth media and seeded on uncoated 100 × 20 mm of tissue culture dishes (BD bioscience) at 37°C, 5% CO 2 , 5% O 2 in a humidified incubator. .. Growth media contained high glucose Dulbecco's Modified Eagle's Medium (DMEM) supplemented with 10% USDA Tested fetal bovine serum, 100 units/mL Penicillin, 100 μ g/mL Streptomycin (all from HyClone), 10 ng/mL recombinant mouse VEGF (R&D, Minneapolis, MN) and 20 ng/mL Single Cell:Article Title: Posttreatment with dexmedetomidine aggravates LPS-induced myocardial dysfunction partly via activating cardiac endothelial α 2A -AR in mice. Article Snippet: Background: Dexmedetomidine (DEX) administered before or at 30 min after sepsis induction was reported to alleviate septic cardiomyopathy in experimental models.. However, sepsis is a life-threatening organ dysfunction due to infection-induced dysregulated host response, whether DEX treatment in the presence of organ dysfunction affects septic cardiomyopathy is unknown.. This study investigated the effect of DEX posttreatment on septic cardiomyopathy. Cell Culture:Article Title: Posttreatment with dexmedetomidine aggravates LPS-induced myocardial dysfunction partly via activating cardiac endothelial α 2A -AR in mice. Article Snippet: Background: Dexmedetomidine (DEX) administered before or at 30 min after sepsis induction was reported to alleviate septic cardiomyopathy in experimental models.. However, sepsis is a life-threatening organ dysfunction due to infection-induced dysregulated host response, whether DEX treatment in the presence of organ dysfunction affects septic cardiomyopathy is unknown.. This study investigated the effect of DEX posttreatment on septic cardiomyopathy. |
